Lately, AGEs have been considered to play an essential position in aging. The accumulation of AGEs in the pores and skin might be decided by measuring pores and skin autofluorescence or SAF, and values are age-adjusted [29-31]. As AGE production requires estrogen, the estrogen stage postmenopause might affect AGEs [32, 43]. The consequences of isoflavones on AGEs, particularly the inhibition of AGE formation, have been reported [33]. Isoflavones act via three mechanisms: 1) prevention of the Maillard response, i.e., prevention of sugar-protein compound formation; 2) inhibition of the oxidation of sugar, fats and amino acids, thereby stopping the formation of carbonyl teams; and 3) prevention of the conversion of Schiff bases into carbonyl groups. As equol is a metabolite of isoflavones, it might exert similar effects as those of isoflavones. Moreover, as equol exerts anti-aging and anti-arteriosclerotic effects on the skin [44], the skin autofluorescence values may be affected.
Methods: Twenty girls who confirmed premenstrual symptoms and had been nonequol producers were enrolled in an open-label, single-arm, clinical research through which they obtained oral SE5-OH for 2 period cycles. The Each day Document of Severity of Issues (DRSP) whole score was evaluated through the intervention cycles. Before taking SE5-OH, fecal samples have been obtained and subjected to terminal restriction fragment length polymorphism evaluation.
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